Sentence examples for mutants was fully from inspiring English sources

The early flowering phenotype of flc-2 mutants was fully rescued in transgenic lines, demonstrating that the fusion proteins are functional.

The axon regrowth defect in zyg-8 lf) zyg-8 lfwas fully rescued by a single copy transgene expressing ZYG-8 driven by a touch neuron specific promutantsindicating a cell-autonomous function.

The H2O2 sensitivity of daf-16 mutants was fully rescued by a transgene expressing a DAF-16a::GFP fusion protein [ 13], confirming the role of daf-16 in conferring H2O2 resistance.

In contrast, only one of the mutants was fully sensitive to ASP3026 (NA S1206Y) while there were two resistant (L1196M and G1269A) and two highly resistant (L1152R and G1202R) mutants.

As shown in Additional file 4: Figure S3, the morphological phenotype (short aerial hyphae) of the Δ tul- 1, Δ dsc- 2 and Δ sah- 2 mutants was fully complemented by the introduction of the wild-type allele of tul- 1, dsc- 2 and sah- 2, respectively.

Myc-COMMD1 and Myc-COMMD1ΔCOMM mutants were constructed using the Gene-Editor kit (Promega) and the resulting mutants were fully sequenced.

In contrast to the phoP mutant, the two pmr mutants were fully virulent, producing severe diarrhea (Figure 7A), multiplying in the colon, and spreading systemically (Figure 7B).

Moreover, these defective phenotypes could not be found among the rescue flies (Table 4), indicating that the defective phenotypes in the mutants were fully rescued.

Previous studies have reported that the prfA* mutants with mid-level PrfA activity (prfA G155S mutants) were fully virulent when intravenously inoculated into mice based on the bacterial CFU required for a 50% lethal dose (LD50) [32].

Paradoxically, however, IR-, UV-, and mitomycin-C (MMC -sensitive D. radiodurans polA MMC -sensitivelly complementeD.by expradioduransthe polA gene fromutantsR-sensitive Escherichiareoli [4].

In 1994, for example, IR-sensitive D. radiodurans polA mutants were fully complemented by expression of the polA gene from the IR-sensitive E. coli [4]; and in 1996, UV-sensitve D. radiodurans uvrA mutants were complemented by uvrA from E. coli [90], suggesting that these recombination and excision repair genes are necessary but not sufficient to produce extreme DNA damage resistance.