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Exact(8)

The pathogenicity was tested for both wild type Guy11 and Mosdi1 ReGFP mutants, and no significant differences were identified among the strains.

Similar to os9-1, SUBEX-C57Y degradation was also blocked in sel1l mutants and no further increase in protein accumulation was detected in the presence of kifunensine.

In parallel, an increased TAG accumulation rate was observed for the starchless mutants and no substantial decrease in biomass productivity was perceived.

Contrary to MPSS predictions, At1g07930, a gene with very weak expression of 5 TPM in the stamen-less ap3 mutants and no expression within the stamen-enriched sup ap1 mutant was identified within the stamens.

We also determined time-kill kinetics of anti-TB drugs and assessed the emergence of resistant mutants and the concentration range within which resistant mutants and no susceptible mycobacteria were selected.

As expected, the GFP+ cells expressed only the three truncation mutants and no WT BAF57 protein, and importantly, WT BAF57 was restored in GFPlow/- cells, showing that Baf57 ΔR can indeed be converted into the WT allele.

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Similar(52)

However, the distribution was broader than those of ΔN-TClpB and 1E/Q mutant, and no clear border between two states was observed.

The less resistance decrease was observed for the hybridization with mutant, and no decrease was observed with the noncomplementary sequence.

In contrast, only a very low amount of preSak-Myc was detected in the corresponding cell wall fraction of the mutant and no precursor was observed in the protoplasts (Figure 2C).

Immunoblotting with phosphosite-specific eNOS antibodies revealed no Ser phosphorylation in the Δ605 612 mutant, no Ser phosphorylation in the Δ626 634 mutant and no Ser phosphorylation in the Δ14 mutant.

Only a limited reduction in the total chitinolytic activity was detected for the different chitinase mutants, including the quintuple mutant, and no growth or germination defects were observed.

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