Sentence examples for mouse fetal brain from inspiring English sources

The present study was designed to investigate the potential adverse effects of maternal exposure to DCAN on mouse fetal brain.

In conclusion, maternal exposure to DCAN adversely affects mouse fetal brain as evidenced by induction of oxidative stress, apoptotic imbalance and neurodegeneration.

We identified the known interactions in the β-globin locus and the same topological domains in both mouse primary erythroid cells and in mouse fetal brain with the latter having fewer interactions probably due to the inactivity of the locus.

We find that the 'specific' reads represent 61%, 50%, and 88% of total reads ('specific' plus 'non-specific') for mouse primary erythroid cells, mouse fetal brain cells, and HB2, respectively, including the self-ligation and non-digestion events.

However, in mouse fetal brain cells, where β-globin locus is not active, that is not necessary and there are no important elements that need to spatially be in close proximity.

For example each of the β-globin locus experiments was carried out by using one sequencing lane on an Illumina HiSeq machine for each different biological sample (mouse primary erythoid cells and mouse fetal brain cells).

Mouse fetal brains were taken from C57BL/6 wild-type or PARKIN−/− mouse embryos at E15-16.

In-situ hybridization of Kiaa0319 on sections of mouse and human brain indicated that Kiaa0319 is expressed during mouse and human fetal brain development, and is involved in neuronal migration for formation of the cerebral neocortex [ 9].

WDR62 has been shown to be enriched in neural progenitors of the VZ and SVZ of mouse and human fetal brain [ 12].

TET generated 5hmCs in human ES cells and the mouse fetal and adult brain cortex are found almost exclusively in a CG context [ 27, 31].

For example, mice lacking Becn1, the mammalian ortholog of yeast VPS30/ATG6, have abnormal cell proliferation in mammary epithelial ducts and splenic germinal center lymphocytes; mice lacking Ambra1 have increased cell proliferation in fetal brain; and atg7−/− mouse embryonic fibroblast cells have impaired TRP53/p53-mediated TRP53/p53-mediated