Sentence examples for mouse fetal development from inspiring English sources

In general, the expression profiles of imprinted genes are well characterised during human and mouse fetal development, but not in human adults.

In conclusion, our work provides multiple lines of evidence that the non-neural developmental defects in Gad1 and Viaat mutant mice are due to a requirement for GABA signaling in the CNS during mouse fetal development.

NKX6.1, a member of the NK homeodomain transcription factor family, is expressed during mouse fetal development in the majority of pancreatic epithelial cells from the earliest stage of bud formation at E9.5 until the onset of the secondary transition at E13.5 [60].

Wadia et al. (2013) reported that low-dose BPA exposure (250 ng/kg BW/​day) during mouse fetal development altered the composition and organization of the extracellular matrix and accelerated maturation of the presumptive fat pad, an event necessary for ductal invasion and branching.

This study shows that many differences exist between human and mice fetal development.

The importance of IGF-II signaling in mouse fetal growth is well described, with both total and placental specific IGF-II knockout transgenic animals associated with impaired fetal and placental development (9, 10).

We first examined the endogenous pattern of Snail1 expression in the perichondral area of mouse embryos throughout fetal development.

However, post-conception putrescine supplementation in mouse drinking water compromises fetal development, likely due to excess reactive oxygen species produced by placental diamine oxidase oxidizing the exogenous putrescine [ 8].

It is, of course, quite possible that the changes observed in 9-mo-old mouse hippocampi were the result of abnormal neural circuit development in Tg mice during embryonic or fetal development.

Hes1 mutant mice die during late fetal development with defects affecting neural, eye, pancreas, pituitary and lung development, among other organs [22] [26].

It is known that ETNK2 −/− mice often have failed fetal development at the late stages of pregnancy due to extensive placental thrombosis, suggesting a critical role for ETNK2 sequences in hemostasis [20].