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A simple model taking into account the molecular volume of the adsorbent and of water molecules could explain the measured mass loss.
It was suggested that these molecules could explain, at least in part, the link between obesity, insulin resistance, beta-cell dysfunction, endothelial dysfunction, atherosclerosis and cardiovascular disease (CVD) [ 1- 6].
This lack of correct MHC class I molecules could explain why patients of varied genetic backgrounds may have different prognoses upon infection with pandemic (H1N1) 2009 virus or even influenza virus (H5N1).
Concerted evolution of MHC molecules could explain why MHC molecules from separate loci within one species can be like one another without necessarily deriving from a common ancestral locus.
Considering this, the present study was designed to address whether the expression of proteins involved in the resistance to cytotoxic CD8+ T cells such as EPHRIN-A1 and/or SCINDERIN or the downmodulation of HLA-class I molecules, could explain the absence of clinical impact of intratumoral CD8+ T cell infiltration in head and neck cancer.
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This data is consistent with the expected DET reaction between FMD compounds and GSH molecules and could explain the selective cytotoxic effect on cancer cells.
To gain further insight into the structural differences of the two molecules that could explain their difference in localization (and possibly their functionality) we analysed their sequences using the well-established neural network program PONDR [ 43, 44], which gave a convincing prediction of intrinsic disorder for the 300 aa C-terminal part of LdSAcP1 missing in the LdMAcP sequence.
The disappearance of this molecule with time could explain the loss of the stimulating effect observed after 5 h.
54 Further studies on these molecules, NCoR1, MYBL2, and RB1, could explain their role at cellular level and their o be corrected as "interaction with each other molecules in contributing to RB tumorigenesis.
These transient cavities can accommodate small molecules, mimicking protein-ligand recognition, which could explain the selectivity and sensitivity observed for different organic analytes in NMR chemosensing experiments.
Since CD25+ CD4+ T regulatory cells are probably selected in the thymus, specific MHC class II molecules could hinder their development, which could explain the association between HLA and disease susceptibility.
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