Hence recent years have seen human travel data incorporated into models of respiratory disease spread to explain the observed patterns of spatial spread [4], and to inform contingency planning [5], [6], [7].
Protective effects of COX-2 inhibition have also been described in rodent models of respiratory syncytial virus [22] and vesicular stomatosis virus (VSV) [23].
All the models of respiratory symptoms and current asthma were adjusted for the same confounding factors as in the asthma ever models.
Theoretical models of respiratory tract deposition of inhaled particles are compared to experimental studies of deposition patterns in humans and animals, as determined principally by particle size, density, respiratory rate and flow parameters.
With these flow rates near total extracorporeal CO2-removal could be achieved in animal models of respiratory failure, thereby allowing a more lung protective conventional ventilation strategy [ 18, 19].
Theoretical models of respiratory tract deposition of inhaled particles are compared to experimental studies of deposition patterns in humans and animals, as governed principally by particle size, density, respiratory rate and flow parameters.
