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To determine effects of glial scar on OECs migration, we measured the mobility of OECs by using a cell culture model of migration of OECs with co-cultured glial scar tissue in a Boyden chamber.
To quantify the extent of cell migration, we measured the gap of the scratched area at various points and then plotted the average width of the scratched area as a function of time after scratch.
To follow individual cell migration, we measured the distance covered by separate cells at different time points.
To gauge the effect of cell proliferation on promoting cell migration, we measured the growth rate for each cell type.
To assess whether overexpression of C/EBPα is sufficient to reduce cell migration, we measured the migration of HeLa cells using transwell migration assays.
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To understand and address migration variation, we measured the migration distance of a scalar (fluorescently labeled 500 nM OVA*) at a set time and tested significance of unit-to-unit variation with an ANOVA test.
To determine the mechanism underlying FXYD6-induced tumor cell migration and proliferation, we measured the phosphorylation of both kinases when FXYD6 expression levels were modulated.
In order to figure out the mechanism for the inhibitory effect of L2H17 in colon cancer cell migration and invasion, we measured the expression of E-cadherin in L2H17-treated CT26.WT cells.
We measured migration of cups and wear rate.
To quantitate these results, we measured migration rate and directional persistence.
In this prospective cohort study, we measured migration in open-wedge osteotomy in patients following an early full weight bearing protocol and compared the results to those from a historical cohort of open-wedge osteotomy patients who followed a standard protocol (full weight bearing after 6 weeks) using radiostereometry.
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