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We evaluated whether hUCBSC are capable of inhibiting the migration capability of glioma cells both in vitro and in vivo, and whether this effect is mediated by downregulation of the PI3K-Akt pathway.

As expected, overexpression of JAM-A undermined the migration capability of MDA-MB-231 cells, as demonstrated by wound healing and Transwell assays (Fig. 3F and 3G).

Accordingly, the migration capability of hNSCs toward JEG-3 cells was confirmed using an in vitro Trans-well assay, in vivo subcutaneously (s.c).

We supposed that high expression of miR-29a may be associated with migration capability of hepatoma cells.

Upregulation of PTEN resulted in the inhibition of migration capability of glioma cells under in vitro conditions.

Fig. 7A shows that the random migration capability of early passage VSMC from old rats is increased 1.9 fold compared to that of young VSMC (p<0.05).

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The purpose of this study was to investigate the cooperative effects of simvastatin (SIM) and stromal cell-derived factor-1α (SDF-1α) on the osteogenic and migration capabilities of mesenchymal stem cells (MSCs), and construct a cell-free bone tissue engineering system comprising SIM, SDF-1α and scaffold.

Therefore while IL-8 seems to leave T cell stimulation by DC unimpaired, chronic exposure to IL-8 may profoundly affect the migration capabilities of DC towards IL-8 gradients and possibly of other leukocyte subsets as well.

In scratch migration assay, sub-cytotoxic MJ attenuated the migration capabilities of SGC-7901 and MKN-45 cells in a dose- and time-dependent manner.

To further assess the effect of FOXP1 expression on tumorigenicity, we compared anchorage-independent growth rates and migration capabilities of FOXP1-induced with GFP-induced control cells.

The spreading and migration capabilities of HTZ-349 cells were assessed using a scratch (wound) assay measuring the expansion of a cell population on a given surface.

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