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In migration experiments using time-lapse microscopy there was a pronounced and statistically significant decrease in migration of cells treated with CM-SFRP3 compared to cells treated with CM-EV for 12 h (Figure 5D).

Also, using immunofluorescence confocal microscopy there was a lack of labelling for the dominant FMDV cellular receptor, αvβ6 integrin [12], in the GCs (Figure S16).

By light microscopy there was complete absence of inflammatory cells in all types of lesions, including the exuberant proliferating foci.

In a previous study of mice with EAE using electron microscopy, there was no evidence of loss of integrity of the BBB during leukocyte migration [ 21].

As shown using confocal microscopy, there was a co-localization of Sig-1Rs and the 120Q-huntingtin-fragment protein within the cytoplasmic aggregates in the PC6.3 cells (arrow in Figure 1e).

Combining a bioluminescence inhibition assay and two-component fluorescence in order to evaluate cell membrane permeability with atomic force microscopy, there was an absence of detectable bioactivity for GS.

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With high throughput microscopy there is much more data generated than can be annotated or evaluated manually, and so developing a fine-tuned and efficient classification model is paramount for unlocking the potential of high throughput methods.

Under fluorescence microscopy, there were several GFP+ Sk-34-derived cells in the DsRED mouse cardiomyocyte spheres after 6 days of culture (Fig. 1B).

Between microscopy and assisted virtual microscopy there were 87 matches (72.5%) and 33 mismatches (27.5%).

On light microscopy, there were 31 45 glomeruli per level section, of which 1 2 were globally sclerosed.

In the clinics in the east with established microscopy, there were 329/1212 (27.1%) false positive results (χ=2.8, P=0.1).

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