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We can observe that the log2 transformed fold change values are reduced in the microarray data compared to the qPCR.
More recently, there have been further studies that assessed the additional predictive value of microarray data compared to clinical data alone.
For example, the genes RSB1, AZR1, and BAG7 showed a higher level of induction in the microarray data compared to the real-time RT-PCR data.
Furthermore, the environment of repressing LV integrants was enriched in CpG and DNase I sites, as well as in highly expressed genes (based on publicly available microarray data), compared to that of non-repressing LV integrants.
The fact that gene-expression levels of neither CYP19/aromatase nor GATA3 were found to be associated with risk of local recurrences in the two studies [ 23, 24] that gave information about the patients' age could reflect either the lower sensitivity of microarray data compared with RT-PCR or a difference in the studied populations.
Selection of CI resistance CGs were made as follows: 25 top performing genes, 24 genes common to peach and Arabidopsis thaliana (ChillPeach microarray data compared with ColdArrayDB: http://cold.stanford.edu/cgi-bin/data.cgi), and 39 genes unique to peach (i.e. not found in ColdArrayDB).
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The GSEA statistical framework was used as previously described above to evaluate differential expression of the biclusters on our whole-genome microarray data comparing wild type and α9−/− cochlear samples (Table S3).
Microarray data comparing Atnudt7-1 and WT plants indicated transcript levels of several nudix hydrolases were altered.
This expression pattern was similar to our microarray data comparing NPC cells and NP tissues.
The initial statistical analysis of the microarray data comparing the S. aureus control quarters from the different time courses failed to detect any significantly differentially expressed genes.
Previous microarray data comparing differential gene expression in the wild-type and atmyb80 mutant anthers showed a 3.2 fold (p value 0.012) up-regulation of the truncated AtMYB80 transcript in the mutant (unpublished data) [ 3].
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