Sentence examples for mice were twice from inspiring English sources

Groups of 6 SCID mice were twice injected i.p. with 200 µl of immune sera from both pools or with naïve mouse serum, 24 hours apart.

Interestingly, fat pads from Obesity-Resistant mice were twice as heavy as those of Low-Fat mice despite the similar body weights of these two groups.

In the experiment with RLS40 tumor, the mice were twice treated with cyclophosphamide (100 mg/kg), on days 2 and 4 after transplantation; in the experiment with parental RLS tumor, the mice were once treated with cyclophosphamide (200 mg/kg) on day 2 after transplantation.

All mice were twice as likely to initiate syntactic chains in the laboratory than at home (F 1, 82) = 85.73, p < 0.001), and mutant mice in particular initiated approximately 25% more chains than wild-type mice in the laboratory environment (F 1,86) = 17.315, p < 0.001; Figure 4), compared to only 5% more in the home environment.

On day 15 and 16 after tumor cell challenge, mice were twice treated with 100 ul/100 ul pcDNA 3.1 complex with Lipofectamine 2000 in group 1 (control group, n = 5), or with 100 ul/100 ul pcDNA 3.1-ANT1 complex with Lipofectamine 2000 in group2 (therapy group, n = 5) via intratumoral injection.

Many skeletal muscles of Mstn−/− mice are twice the mass of those of Mstn+/+ mice [16] while, in contrast, adipose tissue is greatly reduced in size [17], [18].

The expression level of HSPB8 in tTA/HSPB8 double TG (tTA/HSPB8 TG) mice was twice the NTG level, while expression of the other HSPs tested was unchanged (Figure 5A and B).

It is remarkable that the expression of some genes (Fkbp5, Irak1, Dlk1, Ddc, and Mllt2h) in KO mice is twice that of the levels in WT mice, while the level of expression of other genes (Mobp, Prodh, Plagl1, Eya2, and S100a9) was 40 50% higher than those of WT values.

Although the average MT weight for Obesity-Resistant mice was twice that of Low-Fat mice, this did not reach statistical significance.

The magnitude of the reduction, however, was much smaller, and the abundance of pGSK3βSer9 and β-catenin proteins in LiCl-treated IGF1RNestin−KO mice was twice as much as that in NaCl-treated IGF1RNestin−KO mice.

Although the acute LD50 of Ro-05-9963 inormalal mice was twice that of MIS, this apparent advantage was offset by peak tumour levels 50% or less of those achieved by equimolar injected doses of MIS.