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Exact(6)

In our study, the enhanced exercise endurance of Myog-deleted mice may involve alternative pathways to the ones described above.

Thus, the shift toward the non-amyloidogenic processing in APPYG/YG mice may involve a role of Tyr682 in trafficking of APP along the secretory pathway.

We propose that attenuated FAA in D1R KO mice may involve three distinct processes; circadian oscillator entrainment, incentive motivation, and metabolic homeostasis.

The mechanism of action in mice may involve damage to tumour vasculature or immunomodulation, and these effects may be species-specific.

Therefore, the prodrug-induced protection in the mice may involve other organs besides the heart and that will be subject to future study.

Analyses of total daily activity, FAA as a proportion of daily activity, body temperature, and striatal clock gene expression suggest that the FAA phenotype of D1R KO mice may involve impaired synchronization of food-entrainable oscillators combined with an alteration in the strategy for maintaining metabolic homeostasis, favoring reduced total daily activity over nocturnal hypothermia.

Similar(54)

Considering our data from HET mice, this discrepancy may involve subtleties of the isoform-specific Snap-25 mini-gene used for transgenic rescue (94).

Thus, the observed reduced osteogenesis in c/ebpβ−/− mice in vivo may involve micro-environmental effects that impinge on osteoblast function.

Together, these results support our hypothesis and demonstrate that the nonadaptive anxiety phenotype in 129P2 mice may indeed involve impaired cognitive control of emotional processes, resulting in impaired behavioural flexibility, for example in response to novelty.

The pathogenesis of microcytosis in S. Typhimurium-infected mice in our studies may involve several mechanisms including iron deficiency [37], [47] or fragmentation of erythrocytes [48] known to occur with both iron deficiency [37] and DIC.

The molecular basis for the observed correlations is not clear; we can speculate that the increase of hepatic ADMA in db/db mice induced by silibinin may involve the system of y+ carriers of the cationic amino acid family, which has been shown to play a relevant role in the clearance of dimethylarginines in the liver [ 32].

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