Sentence examples for mice may help from inspiring English sources
In this review we outline how studies in mice may help filling these knowledge gaps.
Further studies using conditional tissue-specific RPTOR and RICTOR deficient mice may help resolve the issue with respect to the relative contributions of mTORC1 versus mTORC2 signaling and fibrogenesis in an in vivo setting.
Research in mice may help explain something that doctors have noticed in people who are infected with H.I.V.: cocaine use seems to make the disease progress faster and lead to more of the opportunistic infections that are the hallmark of AIDS.
Further studies of the mice may help scientists track down at least a few of the genes that predispose people to addictions.
Studies on mGlu1 and mGlu5 receptor knockout mice may help to clarify the specific contribution of these receptor subtypes to the behavioral outcome of PRS in males and females; however, this awaits the development of the PRS model in mice.
The AKAP5 KO and D36 mice described here provide a valuable tool to dissect the role of this AKAP in dendritic localization of PKA and other AKAP5 binding partners and together with other mutant AKAP mice may help unravel the complex spatial signaling that forms the substrate for synaptic plasticity.
The LGI1 knockout mouse may help understand the function of this secreted neuronal protein.
If mice simply give you an "icky-gross" feeling, then looking at enough of them, or hearing how much someone cares about their pet mouse, may help.
Scientists are reporting that, for the first time, they have made an artificial prion, or misfolded protein, that can, by itself, produce a deadly infectious disease in mice and may help explain the roots of mad cow disease.
A9 SCIENCE/HEALTH Seeking Answers on Mad Cow Scientists report that, for the first time, they have made a misfolded protein that produces an infectious disease in mice and may help explain the roots of mad cow disease.
Some fundamental differences in the histological observations between adbn−/− mice and other DGC-associated muscular dystrophy mouse models may help to explain the lack of a functional deficit in adbn−/− mice.
