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In contrast, LPS-treated mice have lost morphological cortico-medullary distinctions (Figure 1G, right panel).
To determine if skin γδ T cells in obese mice have lost their ability to produce cytokines, we examined TNFα production by γδ T cells located along the wound edge.
While humans have lost only the Msx3 gene, mice have lost Ventx, Argfx, Dprx, Shox, Rax2, LOC647589, Tprx1 and Nanognb.
While the human lineage has lost only the Msx3 gene, mice have lost Ventx, Argfx, Dprx, Shox, Rax2, LOC647589, Tprx1 and Nanognb.
ERα knockout female mice have lost their capacity to develop mammary gland tissue beyond the embryonic and fetal stages despite elevated levels of circulating estrogens (17β-estradiol).
Both Joo and Flood used a bracketing measurement, which averages the highest anesthetic concentration at which mice still retain the righting reflex with the lowest anesthetic concentration at which mice have lost the righting reflex to estimate MACLORR.
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By day 14 post infection the mice had lost about 30% of their body weight.
By 8 dpi, the influenza infected C3−/− mice had lost >25% of their day 0 weights (CA/09 p<0.009, VN/1194 p<0.05) and had to be humanely euthanized.
By 14 days of treatment (28 days post-infection), mice had lost more than 20% of their body weight and were moribund (not shown).
At the EC50 dose for induction, half of the mice were sacrificed while the remaining half continued in the dosing protocol, until all mice had lost their righting reflex.
Next, we wondered whether, despite the modest effect of Atm-deficiency on tumor progression, lung adenomas in Atm-null mice had lost the senescence response characteristic of these tumors [37].
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