Exact(3)
Such a metabolic pattern is however not specific for pulmonary infarction.
This metabolic pattern is characteristic of ischemia (i.e., a simultaneous decrease in tissue oxygenation supply and substrate/glucose) [8 10].
This metabolic pattern is consistent with pathologic data on pulmonary infarction, showing that consolidation in pulmonary infarction is mainly caused by central blood alveolar filling with a peripheral inflammatory reaction including a foamy macrophage margin at the edge of central necrosis [10].
Similar(5)
This metabolic pattern was accompanied with transitory higher lactate/pyruvate ratios, indicative of a less balanced cytosolic redox status [ 2].
In addition, the roles of the different phenotypes of PCOS on the metabolic pattern are still debated (19– 23).
In sepsis, the early stress-metabolic pattern is characterized by a high (nCD64, glucose, TG) - low (TC, HDL, LDL) combination in contrast to the moderate pattern of TBI in which only glucose increases combined with a moderate cholesterol - lipoprotein decrease.
The early sepsis stress-metabolic pattern is characterized by a high (nCD64, glucose, TG) - low (TC, HDL, LDL) combination in contrast to the TBI in which with the exception of an increased glucose, lipoprotein decrease is moderate and nCD64 or TG increases are not evident.
The metabolic patterns were shifted towards more reduced metabolites as reflected by higher butanol-to-acetone ratio (11%) and butanol-to-acid ratio (292%).
Related(20)
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