Sentence examples for memory maintenance in from inspiring English sources

Table 1 displays the brain regions activated during spatial working memory maintenance in both groups.

Rather, different regions and a more diffuse network seem to be engaged during spatial working memory maintenance in schizophrenia.

We investigated the neural correlates of spatial working memory maintenance in schizophrenic patients and healthy controls using two functional neuroimaging methods: fMRI and NIRS.

Thus, GSK-3β does have an important role in memory maintenance in the adult brain, but it may also cause NFT formation in the aged brain.

However, as memory maintenance in specific neuronal systems is modeled, it will be important to incorporate both ongoing and variable synaptic activity, and investigate the effects of both recurrent circuits and inhibitory synapses.

Previous speculation regarding how chelerythrine and the zeta inhibitory peptide (ZIP) disrupt memory maintenance in both Aplysia and rats has focused on the ability of these pharmacological agents to inhibit the phosphorylation of proteins by atypical PKM (Cai et al., 2011; Sacktor, 2011) (Box 1).

In short, one key to the CDI of nucleosome specific histone PTMs associated with genes involved in memory maintenance may lie in the symmetrical double-strand DNA sequence-specific code for nucleosome positioning.

The impairment in the experimental group was not due to impairment of memory maintenance, since animals in the group that received aversive conditioning trials and were injected with fluphenazine one day later and tested one day after injection exhibited no impairment of memory recall [see Additional file 1A, left].

GSK+/− mice lacked the ability to maintain memory after recall, suggesting GSK-3β importantly contributes to memory maintenance after recall in normal brains.

The impairment was not due to impairment of memory maintenance, because animals in the group that received appetitive second-order conditioning trials and were injected with epinastine at 30 min after completion of the second conditioning stage and tested 1 day after injection exhibited a significant level of memory recall [see Additional file 3].

The impairment was again not due to impairment of memory maintenance, because animals in another control group that received appetitive conditioning trials and were injected with epinastine 1 day after conditioning and tested 1 day after injection exhibited no impairment of memory recall [see Additional file 1B, left].