Sentence examples for memory dysfunction in from inspiring English sources

Fig. 4 The effects of MEXX on escape latencies (a), and swimming distance (b) during the training-trial sessions and on swimming times during the probe-trial session (c) in the Morris water maze task on scopolamine induced memory dysfunction in mice.

Interestingly, memory dysfunction in aged rats, associated with hippocampal retinoid hyposignaling, is alleviated by RA-induced normalization of this retinoid signaling pathway [20].

This suggests that very low amounts of Aβ oligomer could induce memory dysfunction in young mice, or, alternatively, a combination of CCL2 and other inflammatory factors elicited by Aβ production (such as reactive oxygen species) may influence memory formation.

Working memory dysfunction in schizophrenia is a prominent neuropsychological deficit and is considered to be a promising endophenotypic marker to better understand the pathology and risk for the disorder [5], [6], [7].

Together, BACE1+/− gene deletion was able to more specifically improve memory dysfunction in 5XFAD mice; however, the memory benefit produced by partial reduction of BACE1 was observed in an age-related manner and disappeared as disease progressed into the more severe phase in 5XFAD mice.

In fact, the early episodic memory dysfunction in alcoholism may be related to glial cell alteration leading to an insufficient support of neurons; or to axonal damage and demyelination, which can result in an anatomical disconnection of the hippocampus from the main source of its neocortical inputs.

The broader literature on the presence or absence of working memory dysfunctions in patients with major depression is overall inconsistent.

The visual memory dysfunction present in the offspring was also observable in the adults who converted to major psychoses, but not in the adult relatives who did not.

We tested if a 24-h maternal separation on PND 9 can cause an adult phenotype characterized by altered levels of activity and anxiety, learning and memory dysfunction, deficits in behavioural flexibility (reversal deficits) as well as changes in number of neurons in the hippocampus and its subregions in the mouse brain.

We focused our study on prolonged MV-induced postoperative memory dysfunction involved in systemic inflammatory and neuroinflammatory responses.

Furthermore, several reports have shown that memory dysfunction occurs in the absence of any overt pathology (Dignam, 1990), suggesting a disruption of more subtle physiological processes (Lee et al., 2005).