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Exact(5)

Two alternative models of kinetics of solute redox reactions such as the model of catalytic reactions mediated by conventional redox-polymers and the thermodynamic model of electrochemical doping of π-conjugated polymers with regard to the kinetics of outer-sphere electron-transfer reactions have been discussed in an attempt to account for the experimental results obtained.

This rules out that the observed effects on synaptic inhibition are mediated by conventional NMDA-dependent plasticity at Schaffer collateral synapses.

It has been demonstrated that the targets of the host-immune response mediated by conventional and nonconventional anti- T. cruzi antibodies are distinct [ 47].

The down-regulation of Tregs was additively mediated by conventional DCs and NKDCs expressing IL-12, IFN- γ, and TNF- α.

We comprehensively demonstrated that, in addition to the classical Tfh-cell differentiation mediated by conventional DC, the promoting effect due to CpG was orchestrated in vivo by antigen presentation and IL-6 secreted by monocyte-derived dendritic cells (DC) as shown in their absence.

Similar(53)

Here we map the nuclear import and export signals of Dp71d by truncation and point mutant analysis, showing for the first time that Dp71d shuttles between the nucleus and cytoplasm mediated by the conventional nuclear transporters, importin (IMP) α/β and the exportin CRM1.

Genetic deficiency of C3, C5, and/or FcRγ did not affect T-cell activation or autoantibody production, indicating that the pro-arthritogenic activity of C5 is mediated by its conventional effector mechanisms (likely C5a production).

Although segregation in trypanosomes appears to be mediated by a conventional microtubule - kinetochore attachment, the number of kinetochores seems to be less than the number of chromosomes [ 35, 36].

Taken together, these results strongly suggest that His C mutant cells complete DNA replication in S phase without inducing significant DNA damage or replication stress and that the cell cycle arrest at the G2/M transition in His C mutant cells is not mediated by the conventional S phase checkpoints.

In that case, SIRT1 expression is mediated by a non-conventional signaling mechanism in which Sp1, but not PPRE, has a central role.

The protective benefits of TLR9-induced Type I IFN are thought to be mainly mediated by TLR9 activation in conventional DCs (cDCs), as depletion of cDCs abolished the anti-inflammatory effects of pretreatment with CpG-ODN [31], [40].

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