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The strong inhibition of LDL uptake upon long incubation of the cells with several compounds may reflect secondary effects such as down regulation of LDL receptor expression.

Structural changes in other brain regions may reflect secondary neuropathology due to non-specific toxicity of lactacystin or are the consequence of a functional reorganisation of basal ganglia circuitry following DA depletion.

Contrary, the distribution patterns of Aurelia lineages, which lack significant isolation by distance, suggest peripatric origins from a widespread ancestral population, whereas sympatric occurrences may reflect secondary contacts.

This increased hybridization to the WT4 band, as compared to results with P1, may reflect secondary structure differences between probes P1 and P2.

Interference with other activities, including increased sleep problems such as reported in a recent Canadian study,[ 36] and as reported in this study, may reflect secondary effects of these toxicities.

A relatively higher incidence of both keratoconus and cataract was observed in the CRB1-related group, which may reflect secondary effects of a more severe retinal dystrophy compared to other genes.

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Other research identifying negative associations with wetland size and R. aurora may reflect the secondary effects of exotic predators, i.e., smaller wetlands are less likely to maintain fishes or bullfrogs (Richter and Azous 1995; Adams 1999; Pearl et al. 2005).

It is possible that the increased proliferation may reflect a secondary response to the compromised barrier function of the epidermis.

Such a sub-population was observed in all six repeats of this experiment and may reflect a secondary effect of the mutation.

The intensity of bone mineralization, spread over the prosthetic surface, and spread longitudinally over time may reflect the secondary stabilization of a particular stem.

Instead, direct inhibition of pathological limbic activity in areas such as the amygdala and sgACC may be more essential to correcting the pathophysiology associated with mediating depressive symptoms, and the reduction in orbital cortex/VLPFC activity may reflect a secondary relaxation of these areas, once they are no longer recruited to modulate pathological limbic activity.

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