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Studies have suggested that CMV infection may influence cardiovascular disease (CVD) risk and mortality.
Thus, vitamin D has an important role in glycemic control, which may influence cardiovascular outcomes [ 27].
Changes in the microbial flora are associated with obesity [ 3, 4] and microbial metabolism may influence cardiovascular disease [ 5].
While the exact mechanism by which phthalate exposure may influence cardiovascular health is unknown, phthalates are hypothesized to be endocrine disruptors that affect the action and metabolism of androgens, cortisol, and thyroid hormones [ 6– 8].
The clinical relevance of these observed differences in cardiac autonomic function among ART regimens and how they may influence cardiovascular outcomes in HIV-infected individuals need to be investigated.
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As traffic-related air pollution may impact cognition through effects on cardiovascular health or through mechanisms which may also influence cardiovascular outcomes, we hypothesized that HFE polymorphisms would also modify a previously observed association between traffic-related air pollution exposure and cognition in older men.
In addition, aspects of the built environment and airborne PM may also influence cardiovascular risk indirectly, through more proximate factors such as physical activity, social integration and social supports, and exposure to chronic stressors (arrow 5).
Pathogenic M3R autoantibodies may also potentially influence cardiovascular autonomic responses.
Despite these beneficial effects, metformin presents potential disadvantages that may influence the cardiovascular system.
Both macronutrients and micronutrients intake related to a Western diet may influence fetal cardiovascular and metabolic development, through influences on placental transfer and subsequently offspring fat deposition, adipocyte function, pancreatic function and food preference [ 91, 94].
A number of observations suggest that GLP-1 may influence the cardiovascular system [ 14] and exert direct cardiac protective actions through the interaction with its receptor localized in cardiac cells [ 15, 16].
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