Sentence examples for mapping studies revealed from inspiring English sources

Early mapping studies revealed that Oct4, Sox2 and Nanog co-bind gene promoters of many mESC and hESC genes [ 23, 24].

Nucleosome mapping studies revealed global effects of RSC and ISW1a complexes acting against each other in shaping the promoter chromatin architecture.

Genetic mapping studies revealed clusters of congruent or overlapping QTL on LG 2 and 17 that control eye size, VAB and the number of EO SN.

Our mapping studies revealed over 18,000 Alu-DR2 elements lying within -10 kb and +10 kb of the 5'ends of >10,000 genes, [see Additional file 2], representing a substantial proportion of human genes.

In total, the mapping studies revealed five QTLs on chromosomes 2, 5, distal 16, 17 and 19 (Qivr5-2, Qivr16 Qivr16, Qivr17-2 and Qivr19) that did merit further analysis because they were consistently observed in at least two traits and exerted an effect on at least two different days p.i.

Mapping studies revealed that the N-terminus of L-terminase ATPase domain (residues 1 58) contains a minimal S-terminase binding domain sufficient for stoichiometric association with residues 140 162 of S-terminase, the L-terminase binding domain.

Such mapping studies reveal that genetic associations with gene expression are common, that they often have large effect sizes, and that regulatory variants act locally and at a distance to modulate a range of regulatory epigenetic processes, often in a highly context-specific manner [ 5].

An epitope mapping study revealed that several citrulline residues were recognized by anti-citBiP antibodies.

Furthermore, fate-mapping studies revealed migration of airway epithelial cells into the sub-epithelial regions of the airway wall.

Results from recent eQTL mapping studies have revealed substantial heritable variation in gene expression within and between populations.

Epitope mapping studies have revealed that positively charged amino acid clusters with common epitopes of Aβ (HHQKL) and APO-E (LRKRL) are regions that are known to be heparin-binding domains [15].