Sentence examples for defects suggested from inspiring English sources

Our biochemical studies and the Wnt-like appearance of oto defects suggested that altered Wnt signaling is involved in the oto phenotype.

The fact that bub-1 mutants caused only postembryonic-born VD neuron defects suggested two possible reasons: 1) bub-1 is a maternal gene and 2) bub-1 is specifically required at postembryonic stages.

Reduced Mps1 kinase activity allows weakened mitotic checkpoint signaling as well as lagging chromosomes at anaphase, two major defects suggested to contribute to CIN in tumor cells [5], [10].

Overexpressing LARGE can bypass hypoglycosylation of α-DG caused by non LARGE defects, suggested that overexpression of LARGE could be a potential therapeutic strategy for patients with hypoglycosylation of α-DG.

Together, these defects suggested disruption of planar cell polarity, which is known to regulate node, kidney and cochlea development.

The absence of beads (= equals the processing defects) suggested the appropriate solvent and process parameter selection for the present medicated nanofibrous system.

However, our results for the T557A mutant, which is defective in Plk1-binding and phosphorylation but does not show any localization defects, suggest that regulation of GCP-WD by Plk1 is not essential for spindle localization of γTuRCs.

Individual deletions of these RL proteins cause no detectable defects, suggesting a dominant role of RHD3 (Zhang et al., 2013).

The characteristic macroscopic lengthscale of these defects suggests that the crystals grow by attachment of single molecules to the nuclei.

The positive MR observed in our graphene samples with fewer defects suggests the presence of weak anti-localization effect.

The observation of the NBE emission peak and weak green emission related to defects suggest high optical quality of the ZnO nanorods grown on the graphene layers.