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In vivo graftings into osteochondral defects demonstrated that our biofabricated porous material is highly biocompatible with cartilage and bone tissue.
The inclusion criteria were knee pathology, chondral defects demonstrated by magnetic resonance imaging (MRI) and/or arthroscopy, injuries of 1.5 8 cm2, no knee instability (anterior, posterior, or lateral), and no injuries on "mirror".
Extensive k-ART simulations using this potential provide complete details of the energy landscape associated with these defects, demonstrated a complex set of mechanisms available to both vacancies and self-interstitials even in a simple environment such as crystalline Ni.
Studies with primary human osteoblast cultures confirmed the bioactivity of these scaffolds, and regeneration of rabbit radial critical defects demonstrated that this material induces new bone defect bridging, with clear evidence of regeneration of original radial architecture and bone marrow environment.
Injection of synthetic RNA encoding Hipk1 into the DMZ resulted in severe gastrulation and neural tube closure defects, demonstrated by a failure to close the blastopore and to fuse the neural tube (Fig. 5A panel a).
All of the bone defects demonstrated complete healing at an average time of 6 months.
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In fact, the most oxide defects demonstrate such PL behavior in the 80 to 300 K range.
These phase excitations (soliton-like topological defects) demonstrate rich and unexpected properties in a magnetic field, due to their magnetic moments.
The fact that the Au atoms only precipitate on deformation-induced defects demonstrates that solute gold atoms act as efficient self-healing agents in the ferrous matrix.
Furthermore, this indicates that the dysfunction is independent of the particular gene resulting in human MADD, as both ETF and ETFDH defects demonstrate similar abnormalities in these studies.
Moreover, ADAMTS knockout mice show profound developmental defects demonstrating their unquestionable importance during vertebrate embryonic development.
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