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Fundoscopy showed a pale edematous optic nerve head surrounded with intraretinal hemorrhages and yellow retinal infiltrates.
Fundus examination revealed a pale optic disc edema surrounded with round intraretinal hemorrhages and round yellow retinal infiltrates.
Fundoscopic examination demonstrated a focal yellow retinal lesion inferotemporal to the optic nerve in the left eye.
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For example, in eye disorders such as Oguchi disease, retinaldehyde fluxes are thought to be high enough to turn the retina yellow, although retinal degeneration does not occur.
Yellow-white retinal lesions with granular border were observed.
It appears as a yellow-white retinal mass that may exhibit an endophytic or exophytic growth pattern.
In recovered patients, hemorrhage and exudation were disappeared completely and yellow-white retinal scars were formed in 10 eyes.
The white cotton wool patches identified in 3 eyes during the initial diagnosis had evolved into large area of yellow-white retinal lesions two months later without treatment of anti-CMV therapy.
Age-related macular degeneration (AMD) is the leading cause of blindness in the developed world.[1] The hallmark of AMD is the progressive accumulation of drusen (sub-retinal yellow deposits) at the macula, that portion of the central retina specialized for fine visual tasks.
EDI-OCT vertical line scans passing through another region of active choroiditis lesions in the periphery shows multiple RPE bumps and elevations (yellow arrows) (b) and outer retinal hyperreflective deposits seen in the macular region (dotted square) (c).
To obtain retinal tissue containing yellow fluorescent protein (YFP -labeled ganglion cells, the Sez-6 knockout line was crossed with the ThYFP -labeledH [11]. Rats were sedated usinganglionne hydrocellside (50 mg/kg body weightheand xylazine (5 mg/kg), and lethally injected with an overdose of sodium pentobarbital (60 mg/knockout
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