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We conjecture that for yeast, formation of solid stress granules is an effective way of preserving the pre-stress state of the cytosol and that a selected set of yeast proteins and RNAs has been modified by evolution to aggregate upon stress.
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Using yeast species, formation of cadmium sulfide (Dameron et al. 1989), lead sulfide (Seshadri et al. 2011), titanium dioxide (Jha et al. 2009a) and antimony oxide (Jha et al. 2009b) nanoparticles are also reported.
Recent data suggest that in yeast the formation of the 18S rRNA 3' end is mediated by the PIN-domain protein Nob1p through an endonucleolytic cleavage event [22] [24].
In fission yeast, while formation of the contractile ring was completed at 30 min, contraction of the ring was not started until 37 min after the onset of spindle pole body separation [64].
In budding yeast, SC formation by Zip1 polymerization may start preferentially from these sites.
In yeast, eccDNA formation requires chromosomal replication as it originates from stalled replication forks [ 31].
Thus, N. castellii CENs are able to facilitate minichromosome propagation and yeast colony formation, presumably by promoting mitotic segregation.
Membrane-cytoplasmic shuttling has been considered in a simulation of the distribution of activated Cdc42 during the early phase of yeast bud formation [ 23].
Much of the machinery responsible for yeast bud formation is shared also by species capable of true invasive hyphal growth (reviewed e.g. in [ 2- 4]).
In the original submission, we described a physiological, early replicative aging-associated event in budding yeast: the formation of a singular and durable protein deposit harboring amyloid-like proteins, which is faithfully partitioned to the mother cell during cell division.
An intriguing new study in this issue of EMBO Journal demonstrates that H3 lysine-4 trimethylation is critical in budding yeast for formation of the programmed DNA double-strand breaks that initiate homologous recombination during meiosis.
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