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*Prevalence for age group 55-59 years was estimated using data for 57-59 agroupoup.
Cumulative TEQ intake from 6 to 25 years was estimated using national food consumption and contamination data of PCB- and dioxin-TEQ intake.
In addition, in 34 female participants stability of DNA-M in blood between 10 and 18 years was estimated using intraclass correlation coefficients (ICCs).
Cumulative TEQ intake from 1 to 5 years was estimated using the PCB- and dioxin-TEQ intake measured with the food questionnaire.
The cumulative risk by birth cohort of developing a cancer over the age range 0-74 years was estimated using an age-cohort model.
Prevalence of individual metabolic component in these children aged below 10 years was estimated using the IDF definition for children aged 10 to 16 years and the modified ATPIII definitions, respectively.
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Time dependent receiver operator characteristic (ROC) curves and area under the curve (AUC) to predict distant metastases within 5 years and 10 years and death within 10 years were estimated using the method described by Heagerty [ 31] with nearest neighbor estimation of the bivariate distribution of time and continuous MS [ 32].
The means of the flowering dates over years were estimated using best linear unbiased prediction with SAS PROC MIXED.
Individual growth trajectories from 0 to 10 years were estimated using mixed-effects linear spline models and differences in trajectories by individual SNPs and allelic score were determined.
Age of onset and projected lifetime risk as of age 75 years were estimated using the two-part actuarial method implemented in SAS version 8.2.
Notes (1) Model-predicted cases and deaths in women aged 0-84 yeare arestimateded using the resident Australian female population in 2007.
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