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Intraocular pressure would be predicted to improve as the load of pigment in the trabecular meshwork subsides.
All of these effects would be predicted to activate SF-1 further [40], [41], [42], [43].
Conversely, Rm2 has high basal signaling and would be predicted to reduce body weight.
These two functions would be predicted to have a pro-atherosclerotic effect.
In aggregate, these activities would be predicted to be broadly anti-inflammatory.
Logically, a decrease in retrograde trafficking would be predicted to result in an expanded Golgi.
Peak levels would be predicted to exceed the maximum safe concentration by about 3.5 fold.
EGFP expression was robust and would be predicted to be extinguished during reprogramming to hiPSC.
This implies that strains with higher potency would be predicted to benefit from being able to resist replacement by a larger collection of actinomycetes, while strains able to resist inhibition would be predicted to be better at invading new colonies.
Decreased ADAR2 function would be predicted to increase nuclear dsRNA.
This would be predicted to result in premature protein termination.
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CEO of Professional Science Editing for Scientists @ prosciediting.com