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The second approach would be population replacement, the primary aim of which is a conversion from transmission-competent to a transmission-impaired organism (Gantz et al. 2015a).
Because infection was acquired in the community, the appropriate denominator would be population based.
In many cases, the sensitivity and specificity would be population and/or family specific.
An alternative approach would be population PK analysis, which could combine information from subjects with intensive and sparse PK sampling and test for multiple covariates on multiple PK parameters simultaneously.
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In our case, the healthy population would be the population free from G6PD mutations.
The analog of an action potential in a population model would be a population burst.
The CAM population would represent an undisturbed population of the pathogen from natural ecosystems, while the CAP and EU populations would be evolved populations in contact with the agro-ecosystem.
This population represents an asymptomatic preclinical OA population within the spectrum of disease development and would be the population in which primary prevention measures would be used.
The corresponding remaining cows for each reference population would be the test population.
Therefore, we assumed that these four geographical areas would be operational populations.
Persons in these positions do not routinely undergo TST screening and would be in populations with unknown TST conversion rates.
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