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Differential counts for neutrophils and eosinophils increased with cumulative WTC workday exposure intensity (dichotomized to < 10 or 10 WTC workdays) (Table 2).
This 24-hr limit is equivalent to a 75-dBA 8-hr workday exposure, with no noise exposure (i.e., noise < 70 dBA) during the remaining 16 hr.
In both our previous investigation and the patrol trooper study the timing of HRV response occurs during or immediately post-exposure; unlike the current study, where HRV was measured in the evening following workday exposure.
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During the day, when exposure and outcome are being measured over the same time period, the potential for confounding bias is greater than at night when there is a lag between workday exposures and nighttime HRV.
The Hadnot Point distribution system had a median TCE monthly average level of 446 μg/L during January 1980 - February 1985(see Table 2), thus resulting in a possible daily exposure as high as 3.6 mg/day, i.e., within the range of workday exposures that occurred in some occupational settings.
Furthermore, we found an inverse dose response relationship between workday PM2.5 exposure and 24-hr, day and night HRV.
Workers reporting no respirator use had a larger decline in HRV with workday PM2.5 exposure; however, the difference was not statistically significant.
This panel study demonstrated decreases in long-duration HRV subsequent to metal-rich workday particulate exposure in a relatively healthy cohort of boilermaker construction workers.
We used linear regression to assess the associations between HRV and workday particulate exposures.
First, the association between workday particulate exposures and nighttime HRV may capture the biologically relevant time course of the response.
dWorked close to these areas for more than half of the normal workdays during exposure period of October 12 21 , 2001
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