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Five weeks after birth, APCMin/+ mice were placed in 10% oxygen and their intestines assessed for polyp formation after 14 wk (age ∼19 wk).
Mice at 6-8 wk age were immunized in the hind footpad with 0.1 ml of 1 1 emulsion of CFA (Difco) and 1 mg/ml KLH (Sigma-Aldrich).
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Cluster 1 showed 407 genes with low expression (35% of total 1171 genes) in 9 V/null and 4L lung across the 4- to 28-wk age range when compared with the WT lung.
Interestingly, G93A mice administered with 25×106 cells survived until 25 wks of age (22 wks of age 25%, 23 wks of age 16.7%, 25 wks of age 8.3%).
Mice treated with Ad5-IL17A or Ad5-LacZ at either 7 wks or 16 wks of age were euthanized at 26 and 27 wks of age, that is, 19 wks and 11 wks post-treatment, respectively.
Body weight, daily body weight gain and feed consumption reduced in quails exposed to heat stress (33 °C) during 2 4 wk of age, but not during 4 6 wk of age and during total period (2 6 wk of age).
Mice were used at 6 12 wk of age, and were sex-and age-matched within experiments as closely as possible.
The experimental period (13 to 29 wk of age) was preceded by an adaptation period (3 to 12 wk of age).
In contrast, tubules with many vacuoles were common in WSB/EiJ mice at 3 wk of age when round spermatids begin to appear and at 5−6 wk of age when elongating spermatids are evident.
As mentioned previously, salivary glands were cannulated with Ad5-IL17A vector at either 7 wks or 16 wks of age.
Severely cachectic mice had decreased wheel running performance at 15 wk of age, while anemia and body weight loss were not present until 18 wk of age.
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