To evaluate this possibility, the SEDDS formulations were dosed along with cyclosporine that further enhanced the bioavailability of paclitaxel when compared with Taxol®.
Functionalization with YIGSR further enhanced lipid synthesis within differentiating adipocytes.
Co-culture with glia further enhanced the transfection efficiency of neurons to almost 60%.
Pre-annealing the naked CdSe film at 350 °C, followed by coating with MnPyP/Polysil further enhanced the electrode PEC characteristics.
Treatment of cell with resveratrol further enhanced the effects of LY-294002 or AKT inhibitor IV on the expression of Bim, TRAIL, DR4 and DR5.
Subsequent treatment of LBs with Na2CO3 further enhanced purity: sodium carbonate stripping greatly reduced the content of actin (Figure 1C) and Clic5 (not shown) in the LB and limiting membrane fractions with consequent enrichment in the stripping buffer.
Co-treatment of EBSS with CQ further enhanced ROS generation.
Conversely, Si-Control cells overexpressed with Hsp27 further enhanced its proliferation and migratory behavior.
At the level of target modulation, silencing AGR2 in combination with antiestrogens further enhanced ER and cyclin D1 downregulation.
Again, a small number of genes (14) had increased expression in B16 vs. melan-a and treatment of B16 cells with RA further enhanced their expression.
UV radiation increased p53 expression in human skin cells, while concurrent treatment with 1,25(OH)2D further enhanced this effect several fold, at 3 and 6 h after UV radiation.
