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Reversion of costly drug resistance mutations has been associated with a rise in viral load and clinical progression [26], and therefore a precedent exists to potentially explain the rise in viral load associated with the onset of AIDS.
In the acute phase of infection, control of HIV-1 and SIV viremia has been correlated with the appearance of virus specific CD8+ T cells [2] [5] and depletion of CD8+ T cells during the chronic phase of SIV infection was associated with a rise in viral load, implicating the importance of CD8+ T cells in controlling SIV replication [5].
In other cases, an increase in the population frequency of escape-mutant viruses coincided with a rise in total viral load (Fig. 6C, SUMA d20-d41).
The result showed that the MVRs of the composites increased nonlinearly with a rise in temperature and an increase of load, but decreased linearly with increasing the filler weight fraction.
The systolic [Ca2+] response correlated with a rise in [Na+]i and sarcoplasmic reticulum Ca2+ load, and was blocked by the NHE1 inhibitor cariporide (CO2/HCO3−-free media).
Transportation energy becomes increasingly important with a rise in population.
Some researchers declared exceptional rise in viscosity with a rise in volume concentration.
Rise in plasma viscosity is associated with a rise in plasma proteins and particularly fibrinogen [ 4].
Unexpectedly, infusion was followed by a decline in circulating CD4+ T cells and a rise in viral load.
Up to fifty percent of patients with essential hypertension are salt-sensitive, as manifested by a rise in blood pressure with salt loading.
Stable isotope methodology revealed that >80% of the propionate load from the inulin-propionate ester is released in the colon coincident with or after a rise in breath H2.
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