However, this would be informative because, although theoretical models assume a constant change in mortality at a given age or across a window of ages [1], [2], [4], genetic diseases show complex patterns of variability in age of onset.
The discrepancy in these results could be due to differences in the sensitivity of the strain used to BPA exposure [ 48], dose selection, route of exposure, time window of exposure (age) and duration of exposure [ 49, 50].
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As a result, what seemed spontaneous, a moment of light falling on a letter and taking us through the window of the ages, began to look irredeemably staged.
We used a critical window of gestational ages 6 16 weeks to evaluate exposures that could have affected urogenital tract development.
Examining whether there is an asymptote to the negative effect of delaying implantation, in addition to determining a window of implant ages of maximum effectiveness, would provide valuable clinical information.
Assessment of skeletal architecture showed that depletion of serum IGF-1 at the beginning of the late growth phase window (4 weeks of age) resulted in significant reductions in Tb.Th and Tb.Sp while Tb.N increased.
When the growth rate of ctsk−/− was compared to that of the wild type animals (WT), we could establish a time window (5 8 weeks of age) within which ctsk−/−display significantly lower body weight and WAT size as compared to WT.
Importantly, the relationship between GP T1 and GA appears minimally affected by postnatal age within a 2-month window of term-equivalent age.
The next step was matching for age, using a window of 5 years.
