These observations together are one possible explanation for why we observed an association between ERI and isofurans, but not F2-isoprostanes.
This may explain why we observed that the 3′-UTR CRP rs1130864 A minor allele carrier state is associated with autoimmune thyroiditis stringently in female patients.
This finding also explains why we observed that the communities extracted from the multiplex network are mainly pivoted on SMS communities.
This also provides an explanation as to why we observed no difference in either the transcriptome or metabolome profiles between the two test and the control groups.
This correlation also contributes to explaining why we observed increased GMD in the ACC and anterior insula in ETTH not like that most studies reported decreases here in chronic pain diseases, including chronic TTH [16, 35].
This would explain why we observed no increase in neurogenesis in the HD hippocampus in vivo.
The reasons why we observed conflicting results between species for the effect of age on sperm robustness remain unclear.
Therefore, it is explainable why we observed higher excess P&I mortality during pre-SARS vaccine-mismatches than post-SARS.
However, this does not explain why we observed this significant increase in chromatin condensation following 6 h incubation.
That's why we observed a delay in osteoclast formation by 5 to 6 days with the bone slices.
This may explain why we observed that chylomicron formation promoted the delivery of dietary OVA to adipose tissues.
