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Also, approximately 170° kinks are mainly caused by small-angle boundaries, where the insertion of extra atomic planes could make the NWs slightly bent.
This is the case where the insertion of Tos17 in the upstream of a gene caused the repression of downstream gene expression.
However, the approximately 90° kinks result from the local amorphorization of InP NWs while the approximately 170° kinks are mainly caused by small-angle boundaries, where the insertion of extra atomic planes could make the NWs slightly bend.
Palpation is performed by placing the distal phalynx of the index finger in the lower one-third of the vagina, as this is the site where the insertion of the pubovisceral muscle to the pubis is expected (Fig. 4) [25].
Perfect segments, unambiguously identified as 3C-SiC, alternate with defective regions, where the insertion of planar defects and in particular stacking faults on (111) planes perpendicular to the growth axis are found.
However, for approximately 90° bending, local amorphorization is believed to be the main cause for this phenomenon while approximately 170° kinks are mostly induced by small-angle boundaries, where the insertion of extra atomic planes could make the NWs slightly bent.
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Proof Proof of Lemma 3.8 To prove the lemma, we define a homotopy contraction h A : B ¯ U A → B ¯ U A as ∑ m = 1 ∞ 1 ⊗ m ⊗ η where η is the insertion of the identity.
Thus, if ϕ ∈ F 1 Hom M o d ∞ (A, M ) is a homomorphism, then ∂ (b ϕ h A ) = b ϕ ∂ h A = b ϕ implying that it is a boundary and therefore F 1 Hom (U A, M ) = 0. □ To prove the lemma, we define a homotopy contraction h A : B ¯ U A → B ¯ U A as ∑ m = 1 ∞ 1 ⊗ m ⊗ η where η is the insertion of the identity.
Of the 325 clones where the insertion site was mapped, 50% of the insertion sites were found within chromosome 5, and of these more than 40% were within 1 Mb of the site of origin (summarised in supplementary material Table S2).
In cases where the insertion could lie on either of the homologs the first digit of the contig number is replaced by an x.
One of these sets, where the insertion was between bases 237 and 238 of vraR gave positive results in a diagnostic PCR, with the gene-disrupted strain giving a PCR product 900 bp larger than that of the parental strain.
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