Exact(1)
In doing so, we propose a sequence of events where deficient lipid processing by Sucnr1−/− RPE instigates subretinal accumulation of microglia.
Similar(59)
This is particularly important in the case of intellectual capital, where a deficient accounting framework for intangibles leads stakeholders to seek enlightenment beyond the financial statements.
The underlying systems are more general, where the deficient transition descriptions are assumed to be completely known, polytopic uncertain, partly unknown, and completely unknown transition rates.
It will be necessary to consider the direct link between Nck and ADAP in experiments where SLP-76 deficells cells or ADAP mutants lacking the YDDV motifs are analyzed.
Our results extend to human tissue the role of SepA described in rabbit, where SepA deficient S. flexneri significantly reduced the ability of S. flexneri to induce mucosal atrophy and tissue inflammation [11].
These results were similar to those seen for the gelatinases where mice deficient for MMP-2 or MMP-9 or both MMP-2 and MMP-9 do not have a renal phenotype, although in vitro organ cultures did demonstrate that MMP-9 might play a role in ureteric bud branching [19], [20], [21], [22].
There were no significant differences in either signalment or cobalamin status (where cobalamin deficient or not) between the dogs on the test and control treatments.
The exception to this is DGAT, where mice deficient in DGAT-2 die shortly after birth, in part due to an abnormality in permeability barrier function.
Randomised or quasi-randomised controlled trials: where G6PD deficient individuals are allocated to treatment with PQ /8AQ or without PQ /8AQ; where individuals with G6PD deficiency are allocated to different doses of PQ /8AQ.
where G6PD deficient individuals are allocated to treatment with PQ /8AQ or without PQ /8AQ; where individuals with G6PD deficiency are allocated to different doses of PQ /8AQ.
Recently, a first in vivo study showed a pro-inflammatory role for miR-155, where miR155 deficient mice were protected against experimental autoimmune encephalomyelitis following defective T-cell responses [ 88].
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