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All included studies were thus assessed for methodological quality using recognised screening criteria [12].
Entropy, signal-to-noise, and covariance variables were, thus, assessed since they quantify the characteristics of response pattern (e.g. the extent of irregularity/heterogeneity [64] [66]) produced by underlying behavioral microvariables, and would therefore be more sensitive to patterns in approach and avoidance.
In total, 21 inflammatory mediators were thus assessed over time in the following experimental scenarios: no treatment (n = 6 mice per group); 1, 2, 3, or 4 h following surgical cannulation trauma (ST; n = 6 mice per group); or 1, 2, 3, or 4 h of ST + HS (bleeding to 25 mmHg; n = 6 mice per group).
Expanded chondrocytes were, thus, assessed by flow cytometry.
Patients were thus assessed routinely every 4 weeks throughout the duration of the study.
Nurses allocated to the training group (TG) were thus assessed before PTP (T1), just after PTP (T2) (3 months after T1) and 3 months later (T3).
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Dendritic behavior was thus assessed by measuring the rate of DC aggregation.
The individual signal is thus assessed by the probability, p, of the deviation in the context of observed signals.
The presence of Ser/Thr pPTEN and pAkt was thus assessed in the cytosolic extract of cells treated for 1 or 3 min with p17 in the presence or in the absence of Y-27632.
The expression of transcripts for Foxp3 by Treg cells and GATA-3 by the Th2 subset was thus assessed in splenocytes of the various groups of mice by RT-qPCR.
The accumulation of Per2 and Bmal1 transcripts, as strongly cycling representatives of the circadian clockwork, was thus assessed in the liver and pituitary gland of sham-operated and adrenalectomized mice sacrificed every 6 hours after 10 days of DRF or NRF (Figure 4).
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