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Such volatile substances of limited size were found to trigger the activation of a pathogenesis-related gene and genes involved in metabolism (Almeras et al. 2003).
The 8- and 12-membered cyclic analogs proved superior to the remaining compounds and were found to trigger apoptotic cell death in DOHH-2 cells.
Four PRBC samples (13.1%) were found to trigger HLEC apoptosis in these conditions (Table 1, Figure 1).
All 3 compounds were found to trigger expression of the metabolic genes CYP3A4, UGT1A1, and MDR1.
Also, individual compounds were found to trigger specific pathways within the nongenomic signaling network leading to different end points.
Some inflammatory stimuli were found to trigger the production of Wnt5a, which subsequently induced the secretion of inflammatory cytokines [ 6- 8].
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In humans, this has been found to trigger a tangling of such proteins in the neurons – a hallmark of Alzheimer's disease.
At both these subduction zones, the slow slip earthquakes have been found to trigger earthquake swarms.
Remarkably, soluble CD4 was found to trigger dramatic enhancement of this virus.
TRPA1 activation by AITC has been found to trigger vasodilatation in rat cerebral arteries in an endothelium-dependent mechanism.
Some of the slow slip earthquakes have also been found to trigger earthquake swarms (Delahaye et al. 2009; Hirose et al. 2014).
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