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During adulthood, animals were bred with drug-naïve colony males.
All donor mares were bred with EAV-infective semen from a stallion persistently infected with the virus.
(I) EIIa-Cre mice were bred with Atg7flox/flox mice, and the progeny heterozygous Atg7+/− mice were intercrossed for the generation of E13.5 homozygous Atg7−/− mice.
Male chimera mice were bred with C57BL/6N female mice.
ATG7flox/flox mice were bred with Lyz-Cre mice.
Homozygous parkin −/− mice were bred with homozygous hA30Pα-syn mice.
CAGGFP-Spry1 female mice were bred with Tie2-Cre male mice to generate conditional transgenic mice.
GM60 mice were bred with VGluT3−/− mice [29] to generate GM60 VGluT3−/− animals.
These offspring were bred with each other to produce animals homozygous for the mutation.
These animals were bred with the tgind β2a as described below.
OT-1 mice were bred with Thy1.1+ mice to generate the Thy1.1+OT-1+ mice.
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