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The obvious increase in the animals' weight was demonstrated to be significantly different between days 0 and 14 and days 7 and 14 with P value of <0.05 as tested by one-way ANOVA.
For RASSF1A, no correlation between methylation levels and serum calcium, PTH or glandular weight was demonstrated; however, increased levels of RASSF1A methylation correlated negatively with serum calcium (Table 2).
Finally, homology in QTL regions between sheep and cattle for muscle, bone, fat and body weight was demonstrated.
The highest mean birth weight was demonstrated for GDM cases in screening regime C (3636 g) (Table 3).
High PHA accumulation of up to 52% dry cell weight was demonstrated in marine cyanobacterium Synechococcus sp. PCC 7002.
In the Hyperglycemia and Adverse Pregnancy Outcome (HAPO) study, a continuous relationship between maternal glucose levels and birth weight was demonstrated, indicating that even moderately elevated glucose levels may increase risk of fetal overgrowth [ 7].
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Growth rates were also sensitive to nonionic surfactant additions where molecular structure and weight were demonstrated to be important variables.
Low maternal folate levels during pregnancy and low birth weight are demonstrated to be associated with vascular endothelial dysfunction in newborn infants [ 37, 118, 119].
In clinical trials, significant reductions in serum glucose and weight were demonstrated for exenatide with primary glycemic effects of the twice daily formulation on prandial glucose control.
Similar findings on the association between maternal smoking and low birth weight are demonstrated in the recent cohort studies conducted in Australia [ 33] and the UK [ 34].
The sex ratio was 1.356 for preterm birth, and it proportionally declined with an increase in the GA to the lowest level at GA ≥41 weeks.> -wrap-foot> * p < 0.05 The sex ratio by birth weight is demonstrated in Fig. 4.
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