All the rats started to show weight bearing on all four limbs after 2 weeks onwards.
Ossification centres, visualised as areas of increased echogenicity, are evaluated with ultrasound from 9 gestational weeks onwards [13, 14, 15].
However, there was a significant (p ≤ 0.05) decline in percentage CP with storage from 24 weeks onwards for all the grass species in the three curing periods.
Paper 3 showed an initial significant difference, but this declined until no longer significant at 18 weeks onwards, after which the SFS used was switched, then showing a significant long-term difference.
While not different at 4 weeks, body weights began to diverge at 8 weeks, with WKYs significantly heavier than GKs from 12 weeks onwards.
From 6 weeks onwards, these rats were fed a high-sodium diet (containing 8% NaCl) or a low-sodium diet (containing 0.3% NaCl) [10], [11].
Minor and large erosions of the gastric mucosa, which are precursor lesions of gastric ulcer, were present in 100% of WT-infected animals from 8 weeks onwards.
Some protection was seen up to six weeks but from seven weeks onwards there was no evidence of any remaining benefit of having received IPTi.
There were no differences between any of the WT groups except in the male handling groups, where the playground mice increased weight significantly compared to the home cage mice, from 22 weeks onwards (p = 0.027; Figure 9E).
The MEPI index will be determined from six weeks onwards.
From 2 weeks onwards, no labeled cells were observed.
