Sentence examples for weak stimulation from inspiring English sources

Whereas one might expect only the strongly stimulated synapse to undergo LTP (since weak stimulation alone is insufficient to induce LTP at either synapse), both synapses will in fact undergo LTP.

Such relatively weak stimulation of the MLR produced locomotor movements after a relatively long delay, which featured neuronal interactions in the hindbrain.

In the regime of linear responses, i.e., for weak stimulation, we used an alternative method of sensitivity estimation [28]: the external force was zero mean broadband Gaussian noise with the standard deviation σ s, band-limited to the cutoff frequency of fc = 200 Hz, Fext(t) = s(t).

But upon simultaneous weak stimulation, both synapses undergo LTP in a cooperative fashion.

; Associativity Associativity refers to the observation that when weak stimulation of a single pathway is insufficient for the induction of LTP, simultaneous strong stimulation of another pathway will induce LTP at both pathways.

The ectopic expression of p53 resulted in a weak stimulation for all reporter constructs (2.0, 2.6 and 2.3 fold, respectively, for empty vector, FLT-C and FLT1-T).

A divergence appears only at weak stimulations (below 10−3 µM/s) where the rising time of RP at soma is shorter than that of SP (Fig. 4C).

Therefore, NMDARs contributed neither to the firing mode nor to the temporal precision of PP inputs in DGCs from chronic epileptic rats when PP-EPSPs were evoked by weak stimulations.

To examine the EPSP spike coupling, weak stimulations were used (see methods) in order to evoke small EPSPs (∼5 mV) at around −50 mV (threshold holding potential), a potential at which EPSPs triggered cell firing in single-spike mode (Fig. 1 A ) in about 50% of the trials (see Table 1) both in DGCs from control and epileptic rats.

Weak stimulations of PP evoked AMPAR-mediated EPSPs (amplitude ∼3 5 mV, see Methods) that triggered cell firing in single-spike mode with a high temporal precision both in DGCs from control and epileptic rats (mean SD = 3.5 ± 0.3 ms, n = 38 cells from control rats; mean SD = 3.1 ± 0.3 ms, n = 23 cells from epileptic rats; P > 0.05; Fig. 6 A, B, D ).

In congruence with the weaker stimulation of iodide flux by cisplatin than by hypotonicity (Fig 3), cisplatin preincubation stimulated taurine efflux much less than hypotonic swelling.