First, we were unable to evaluate dose response interaction as the information was not fully available.
LIMITATIONS: We were unable to evaluate the effect of community care and investigations without hospitalization (office visits, non-invasive testing, etc).
Fourth, due to modeling constraints we were unable to evaluate interaction effects of social support quality and quantity, further research is needed.
Thus, we were unable to evaluate the efficiency of RP703 transcript termination at the predicted site.
Since PC12 cells do not produce epinephrine [16], we were unable to evaluate its secretion.
We were unable to evaluate RP or SP independently due to sample size limitations.
We were unable to evaluate correlations between RSV load in nasopharyngeal specimens and severity, as reported by others [27].
Lastly, we were unable to evaluate why health care providers failed to deliver IPTp for apparently eligible women.
We were unable to evaluate interactions between predictors because of insufficient degrees of freedom (see Table 2 for the models considered, AICC values, and Akaike weights).
Second, we were unable to evaluate the correlation of the non-randomness index with other commonly used sympathetic indicators, such as blood pressure or plasma catecholamine levels.
Therefore, we were unable to evaluate pathologic upstaging data.
