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First we verified, using fUS, that blue light similarly caused consistent macroscopic increases in CBF.
These quantities follow a perfectly linear relationship, as we verified using a light-to-voltage sensor (TSL 235T, TAOS).
Furthermore, we verified using synthetic data that as the number of experimental conditions increases, the network estimation accuracy improves.
We verified using Kaplan Meier curves that simultaneous high MYC and high RAS defined a subtype of poor clinical outcome.
As a first step toward finding tests that were sensitive to familial language impairment, we verified using self-report data that there was indeed a higher prevalence of impairment among the parents of probands with language/literacy impairments.
We hypothesized that some samples had been mislabeled during handling, which we verified using PCR and Sanger sequencing experiments on separate DNA extractions from the source tissues, using primers identified by Galaxy tool #32 to amplify over a subset of the genotyped SNPs.
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We visually verified using an apical 4-chamber view that the RV/LV ratio was < 1.
This has led to statistically relevant and high quality data, which we independently verified using direct transcript level measurements.
We first verified using double immunostainings with anti-EN and anti-Collier (provided by M. Crozatier) antibodies that Collier, even though broadly expressed, overlapped EN expression in row 6 but not in row 7 (data not shown).
We also verified, using the co-IP assay, that STK33 could bind to c-Myc in HKCL2 and SMMC7721 cells (figure 6D).
We then verified, using sub-cellular fractionation, that GFP-FTO expressed in COS7 cells is present both in the nuclear and cytoplasmic fractions, as we had previously observed in MEFs and hypothalamic N46 cells [ 12].
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