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We, therefore, analyzed the bone phenotype of Maged1-deficient mice.
We therefore analyzed the relative contribution of glenoid morphology and version to humeral head position.
We therefore analyzed the clinical characteristics of AM and risk factors for developing migraine later.
We therefore analyzed the morphology of pancreatic islets.
We therefore analyzed cell surface marker expression in the HAK.
We therefore analyzed the structure of the muscle fibers.
We therefore analyzed the expression of NKp30L and NKp44L on the different cell lines.
We therefore analyzed the two subgroups of FTLD-TDP, cases with and without GRN mutations, separately.
We therefore analyzed rgp63-specific production of these antibodies before infection.
We therefore analyzed heparanase levels according to gender (Fig. 2A, B).
In subsequent analyses, we therefore analyzed the position rather than the extent of these activations.
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