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Here we probed if the MBP-TRPV4-Ct can also interact with polymerized microtubules.
Although increased RhoA activity was not detected, we probed if inhibition of downstream effectors of Rho, ROCK-1 and MLCK, could suppress some of morphological features of LT-treated cells.
Next, we probed if HER2 and ObR can physically interact in breast cancer cells.
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We therefore probed if ERK5 could also modulate hsa-miR29b expression (Fig. 3A,B).
We thus probed if pathogenic prions could lead to the phosphorylation of SAPKs.
We next probed if the distal downstream effector of Wnt7a/Fzd9 pathway viz., PPARγ (Winn et al., 2006) could also regulate the expression of hsa-miR29b (Fig. 3E,F).
Since, hsa-miR29b expression was attenuated in all the NSCLC cell lines tested; we probed next if hsa-miR29b could affect NSCLC cell proliferation, either by using gene specific knockdowns (Fig. 2B,C) or re-expression of hsa-miR29b (Fig. 2D,E) in NSCLC cells.
Out of the 170 organisms that we probed, we found that if an organism lacked one of these qualities then it lacked laforin and if it possessed all of these qualities then it possessed laforin [ 15].
When further probed if they would respond to a pandemic "regardless of severity", almost one third (32%) of surveyed staff indicated they are unlikely to do so.
We set out to probe if we could identify direct genetic interactions of loci corresponding to isolated genes, with smn.
If we probe deeper into what is and what is not literature, we find many questions and no solid answers.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com