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We only determined and summed the wavelet coefficients within dyadic timescales ranging from 21 (hourly) to 216 (~4 years) because of the unreliability of the wavelet coefficients associated with longer timescales (217 or ~8 years) as in a previous study (Stoy et al. 2005).
Thus, we only determined complete transection of nerves.
Third, we only determined the change of osmolality after single bolus of hyperosmolar agents.
In a first attempt, we only determined the intensity of the GFP signal.
However, in this study, we only determined the effect of pinocembrin on translocation cascades of MAPKs following the phosphorylation.
In this case, we only determined the presence/absence status for T6SS core components in unassembled contigs.
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For simulations larger than with N = 50 we therefore only determined the theoretical distribution up to a set level of the number of active neurons.
We not only determined the number of surviving cells that are able to form colonies on regular growth media, but also estimated the surviving cells using direct counting techniques including the live/dead stain.
An alternative explanation derives from the fact that we have only determined the average preRC density to be one/100-kb.
We not only determined the total extent of incorporation but also its relation to synthesis of DNA and RNA, which are inhibited by gemcitabine.
Secondly, we not only determined the most influential landscape variables, but also evaluated the predictive power of our models, including the detection of populations that strongly deviate from geographical expectations.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com