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We now test the methods for high-dimensional signals.
We now test the ability of our system in detecting signs of tampering or parcel substitution.
We now test our methods on magnitude spectrograms as described in [47].
We now test our long-term filter banks on both datasets.
We now test whether combined signaling of IL-1 and tumor necrosis factor (TNF) is critical for this process.
We now test NBP, NBP-TG, and NGBP-PJT using real RSS data obtained in indoor environment.
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Using the same experimental design, we now tested the power of retrieval cues to interact with this contextual renewal phenomenon.
We now tested whether tumor cells can activate STAT3 within APC using the contact-dependent mechanism, in addition to soluble factors, and compared these two STAT3 activating pathways.
We now tested the effect of lipids in mitochondria coupled by faf-BSA and GDP, both in the absence and in the presence of Ca2+.
Since we previously observed that unstimulated platelets do not have surface expression of LAIR-1 ([16] and Steevels et al, Manuscript submitted for publication), we now tested whether upon activation platelets LAIR-1 expression was induced.
We now tested the prevalence, clinical, and diagnostic value of YB-1/p18 detection in common tumors.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com