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We next explored other Ca2+-permeable channels involved.
We next explored the identity of the Ca2+-permeable channels.
We next explored the possible involvement of CNG channels.
We next explored the underlying mechanisms for the glucose potentiation.
Klebsiella, we next explored the mechanism accounting for reduced survival.
We next explored the anti-tumorigenic properties of TBX18 and HOXB13 in colon cancer cells.
Thus, we next explored whether loss of the WD40 domain alters LRRK2 complex formation.
We next explored the potential of QM56 to modulate cell injury in vivo.
We next explored the potential mechanisms underlying the inhibitory effects of AICD.
We next explored a possibility that p53 is involved in MIF-induced HIF-1 activation.
We next explored the effect of the presence of NA on HA-mediated fusion.
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