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We next ask the following question: is it possible to build a metric that would predict the effectiveness of a new signature based on its structural characteristics?
Considering that the inter-species modules differ extensively in their composition, we next ask whether these seemingly distinct modules still code some common or even essential biological processes in the genomes of human and mouse.
We next ask the question whether sunitinib might interfere with standard chemotherapeutics applied to glioblastoma patients.
We next ask whether HIRA mediated H3.3 deposition is required for transcriptional reprogramming.
We next ask the question, what is protecting HCT116 p53 −/− cells from apoptosis after the observed severe DNA damage.
We next ask to what extent pleiotropy constrains adaptation of TEM-1 when exposed to both antibiotics simultaneously.
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We next asked whether CRTC2 modulates SREBP1c processing via INSIG2.
We next asked if the three blades were sufficient for secretion.
We next asked whether IKKγΔ was able to bind TANK.
We next asked how the patient-derived rod photoreceptors degenerate.
We next asked whether CSPG also affects ESC-derived nsphs.
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CEO of Professional Science Editing for Scientists @ prosciediting.com